Abstract
Benign prostatic hyperplasia (BPH) and urolithiasis may coexist in older men, particularly when impaired bladder emptying contributes to the development or retention of bladder calculi. However, urinary stasis alone does not fully explain stone formation. Prostatic anatomy and metabolic characteristics may also influence this process. Distinguishing secondary bladder stones from calculi originating in the upper urinary tract is essential because their coexistence with BPH does not necessarily indicate the same causal mechanism.
References
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